[1] |
2015年全国细菌耐药监测报告[J]. 中国执业药师,2016,13(3):3-8.
|
[2] |
GEORGI C,BUERGER J,HILLEN W,et al. Promoter strength driving TetR determines the regulatory properties of Tet-controlled expression systems[J]. PLoS One,2012,7(7):e41620.
|
[3] |
RAMOS J L,MARTÍNEZ-BUENO M,MOLINA-HENARES A J,et al. The TetR family of transcriptional repressors[J]. Microbiol Mol Biol Rev,2005,69(2):326-356.
|
[4] |
FONSECA P,DE LA PEÑA F,PRIETO M A,et al. A role for the regulator PsrA in the polyhydroxyalkanoate metabolism of Pseudomonas putida KT2440[J]. Int J Biol Macromol,2014,71:14-20.
|
[5] |
MILLER R M,TOMARAS A P,BARKER A P,et al. Pseudomonas aeruginosa twitching motility-mediated chemotaxis towards phospholipids and fatty acids:specificity and metabolic requirements[J]. J Bacteriol,2008,190(11):4038-4049.
|
[6] |
KAZAKOV A E,RODIONOV D A,ALM E,et al. Comparative genomics of regulation of fatty acid and branched-chain amino acid utilization in proteobacteria[J]. J Bacteriol,2009,191(1):52-64.
|
[7] |
ZARZYCKI-SIEK J,NORRIS M H,KANG Y,et al. Elucidating the Pseudomonas aeruginosa fatty acid degradation pathway:identification of additional fatty acyl-CoA synthetase homologues[J]. PLoS One,2013,8(5):e64554.
|
[8] |
SUN Z,KANG Y,NORRIS M H,et al.Blocking phosphatidylcholine utilization in Pseudomonas aeruginosa,via mutagenesis of fatty acid,glycerol and choline degradation pathways,confirms the importance of this nutrient source in vivo[J]. PLoS One,2014,9(7):e103778.
|
[9] |
WILLSEY G G,VENTRONE S,SCHUTZ K C,et al. Pulmonary surfactant promotes virulence gene expression and biofilm formation in Klebsiella pneumoniae[J]. Infect Immun,2018,86(7):e00135-18.
|
[10] |
KANG Y,NGUYEN D T,SON M S,et al. The Pseudomonas aeruginosa PsrA responds to long-chain fatty acid signals to regulate the fadBA5 beta-oxidation operon[J]. Microbiology,2008,154(Pt 6):1584-1598.
|
[11] |
YEO H K,PARK Y W,LEE J Y. Structural basis of operator sites recognition and effector binding in the TetR family transcription regulator FadR[J]. Nucleic Acids Res,2017,45(7):4244-4254.
|
[12] |
BYNDLOSS M X,RIVERA-CHÁVEZ F,TSOLIS R M,et al. How bacterial pathogens use type Ⅲ and type Ⅳ secretion systems to facilitate their transmission[J]. Curr Opin Microbiol,2017,35:1-7.
|
[13] |
SHEN D K,FILOPON D,KUHN L,et al. PsrA is a positive transcriptional regulator of the type Ⅲ secretion system in Pseudomonas aeruginosa[J]. Infect Immun,2006,74(2):1121-1129.
|
[14] |
KANG Y,LUNIN V V,SKARINA T,et al. The long-chain fatty acid sensor,PsrA,modulates the expression of rpoS and the type Ⅲ secretion exsCEBA operon in Pseudomonas aeruginosa[J]. Mol Microbiol,2009,73(1):120-136.
|
[15] |
陈思遐,沈自燕,冯旰珠. 铜绿假单胞菌毒力因子的研究进展[J]. 国际呼吸杂志,2018,38(18):1410-1413.
|
[16] |
YIN L M,EDWARDS M A,LI J,et al. Roles of hydrophobicity and charge distribution of cationic antimicrobial peptides in peptide-membrane interactions[J]. J Biol Chem,2012,287(10):7738-7745.
|
[17] |
LATENDORF T,GERSTEL U,WU Z,et al. Cationic intrinsically disordered antimicrobial peptides(CIDAMPs) represent a new paradigm of innate defense with a potential for novel anti-infectives[J]. Sci Rep,2019,9(1):3331.
|
[18] |
GOODERHAM W J,BAINS M,MCPHEE J B,et al. Induction by cationic antimicrobial peptides and involvement in intrinsic polymyxin and antimicrobial peptide resistance,biofilm formation,and swarming motility of PsrA in Pseudomonas aeruginosa[J]. J Bacteriol,2008,190(16):5624-5634.
|
[19] |
HAN M L,ZHU Y,CREEK D J,et al. Alterations of metabolic and lipid profiles in polymyxin-resistant Pseudomonas aeruginosa[J]. Antimicrob Agents Chemother,2018,62(6):e02656-17.
|
[20] |
TRIMBLE M J,MLYNÁRIK P,KOLÁ M,et al. Polymyxin:alternative mechanisms of action and resistance[J]. Cold Spring Harb Perspect Med,2016,6(10):a025288.
|
[21] |
MO C Y,BIRDWELL L D,KOHLI R M. Specificity determinants for autoproteolysis of LexA,a key regulator of bacterial SOS mutagenesis[J]. Biochemistry,2014,53(19):3158-3168.
|
[22] |
LAI B M,YAN H C,WANG M Z,et al. A common evolutionary pathway for maintaining quorum sensing in Pseudomonas aeruginosa[J]. J Microbiol,2018,56(2):83-89.
|
[23] |
NOVOVIC K D,MALESEVIC M J,FILIPIC B V,et al. PsrA regulator connects cell physiology and class 1 integron integrase gene expression through the regulation of lexA gene expression in Pseudomonas spp.[J]. Curr Microbiol,2019,76(3):320-328.
|
[24] |
MA L,CONOVER M,LU H,et al. Assembly and development of the Pseudomonas aeruginosa biofilm matrix[J]. PLoS Pathog,2009,5(3):e1000354.
|
[25] |
LIN J,CHENG J,WANG Y,et al. The Pseudomonas quinolone signal(PQS):not just for quorum sensing anymore[J]. Front Cell Infect Microbiol,2018,8:230.
|
[26] |
WADE D S,CALFEE M W,ROCHA E R,et al. Regulation of Pseudomonas quinolone signal synthesis in Pseudomonas aeruginosa[J]. J Bacteriol,2005,187(13):4372-4380.
|
[27] |
NGUYEN D,JOSHI-DATAR A,LEPINE F,et al. Active starvation responses mediate antibiotic tolerance in biofilms and nutrient-limited bacteria[J]. Science,2011,334(6058):982-986.
|
[28] |
KOHANSKI M A,DWYER D J,WIERZBOWSKI J,et al. Mistranslation of membrane proteins and two-component system activation trigger antibiotic-mediated cell death[J]. Cell,2008,135(4):679-690.
|
[29] |
DREES S L,LI C,PRASETYA F,et al. PqsBC,a condensing enzyme in the biosynthesis of the Pseudomonas aeruginosa quinolone signal:crystal structure,inhibition,and reaction mechanism[J]. J Biol Chem,2016,291(13):6610-6624.
|
[30] |
DULCEY C E,DEKIMPE V,FAUVELLED A,et al. The end of an old hypothesis:the Pseudomonas signaling molecules 4-hydroxy-2-alkylquinolines derive from fatty acids,not 3-ketofatty acids[J]. Chem Biol,2013,20(12):1481-1491.
|
[31] |
WELLS G,PALETHORPE S,PESCI E C. PsrA controls the synthesis of the Pseudomonas aeruginosa quinolone signal via repression of the FadE homolog,PA0506[J]. PLoS One,2017,12(12):e0189331.
|
[32] |
COOKE A C,NELLO A V,ERNST R K,et al. Analysis of Pseudomonas aeruginosa biofilm membrane vesicles supports multiple mechanisms of biogenesis[J]. PLoS One,2019,14(12):e0212275.
|
[33] |
CIOFU O,TOLKER-NIELSEN T. Tolerance and resistance of Pseudomonas aeruginosa biofilms to antimicrobial agents-How P. aeruginosa can escape antibiotics[J]. Front Microbiol,2019,10:913.
|