[1]Goldstein LB,Bushnell CD,Adams RJ,et al.Guidelines for the primary prevention of stroke: a guideline for healthcare professionals from the American Heart Association/American Stroke Association[J].Stroke,2011,42(2):517-584.[2]钟琳玲,廖芳.血清同型半胱氨酸与冠心病的相关性研究[J].实用心脑肺血管病杂志,2011,19(10):1713.[3]Wang JJ,Zhang CN,Meng Y,et al.Elevated concentrations of oxidized lipoprotein(a)are associated with the presence and severity of acute coronary syndromes [J].Clin Chim Acta,2009,408(1-2):79-82.[4]Lippi G,Targher G,Franchini M,et al.Biochemical correlates of lipoprotein(a)in a general adult population.Possible implications for cardiovascular risk assessment [J].J Thromb Thrombolysis,2009,27(1):44-47.[5]Speidl WS,Nikfardjam M,Niessner A,et al.Mild hyperhomocysteinemia is associated with a decreased fibrinolytic activity in patients after ST-elevation myocardial infarction.[J].Thromb Res,2007,119(3):331-336.[6]Ebbing M,Bφnaa KH,Arnesen E,et al.Combined analyses and extended follow-up of two randomized controlled homocysteine-lowering B-vitamin trials[J].J Intern Med,2010,268(4):367-382.[7]Carey VJ,Bishop L,Laranjo N,et al.Contribution of high plasma triglycerides and low high-density lipoprotein cholesterol to residual risk of coronary heart disease after establishment of low-density lipoprotein cholesterol control[J].Am J Cardiol,2010,106(6):757-763.